Wednesday, August 21, 2013

Pathogenesis of Acute Coronary Syndrome part 2

         Recent pathogenesis explains acute coronary syndrome, acute coronary syndrome (ACS) is caused by obstruction and thrombotic occlusion of the coronary arteries, which is caused by a vulnerable atherosclerotic plaque erosion, fissure, or rupture. The main cause of acute coronary syndrome triggered by erosion, fissure, or rupture of atherosclerotic plaques is due to the presence of the condition of unstable atherosclerotic plaques (vulnerable atherosclerotic plaques) with characteristics; substantial lipid core, thin fibrous cups, and the plaque shoulder (shoulder region of the plague) full the activity of inflammatory cells such as T lymphocytes and others (Figure A). Thick plaque that can be seen with the percentage of narrowing of the coronary arteries on coronary angiography examination does not mean anything as long as the plaque in stable condition. In other words, the risk of plaque rupture in atherosclerosis is not determined by the amount of plaque (degree of constriction) but by vulnerability (vulnerability) plaques.

Friday, August 16, 2013

Pathogenesis of Acute Coronary Syndrome part 1

       Acute Coronary Syndrome is one of the clinical manifestations of coronary heart disease is mainly caused by the addition of the further atherothrombotic ischemic stroke, and peripheral arterial disease (PAD). Further atherothrombotic is a chronic disease with a very complex process and multifactorial and interrelated.

Tuesday, August 13, 2013

Clinical Features of Coronary Heart Disease

1. Clinical features of coronary heart disease:
Several days or weeks before the body was not powered, chest discomfort, heart beating fast when sports or move, gasping for breath, sometimes accompanied by nausea, vomiting and body secretes more sweat.

Sunday, August 4, 2013

Coronary Heart Disease Risk Factors

There are factors which can cause or trigger Coronary Heart Disease:
1. Lipid
        Dyslipidemia believed to be the major risk factors that can be modified for the development and progressive changes in the occurrence of coronary heart disease. Cholesterol is transported in the blood in the form of lipoproteins, 75% is a low-density lipoprotein (low density liproprotein / LDL) and 20% is a high density lipoproteins (high density liproprotein / HDL). HDL-cholesterol was lower has a good role in coronary heart disease and there is an inverse relationship between HDL levels and the incidence of coronary heart disease.

This Blog Contents are protected by